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Δευτέρα 16 Οκτωβρίου 2017
Visual and Postural Motion-Evoked Dizziness Symptoms Are Predominant in Vestibular Migraine Patients.
Visual and Postural Motion-Evoked Dizziness Symptoms Are Predominant in Vestibular Migraine Patients.
Pain Med. 2017 Jul 13;:
Authors: Vuralli D, Yildirim F, Akcali DT, Ilhan MN, Goksu N, Bolay H
Abstract
Background: Vestibular migraine (VM) is one of the most common underdiagnosed disorders. We aimed to study the clinical characteristics of VM patients who were referred to a neurology-headache unit by otolaryngology after exclusion of peripheral causes of vertigo.
Methods: One hundred and one patients diagnosed with VM in the headache unit were included. Description of vestibular symptoms, demographic and clinical features, trigger factors, accompanying diseases, and response to vestibular-suppressant medications and prophylactic migraine treatment were evaluated.
Results: Vestibular symptoms were triggered by daily head and body movements and mainly consisted of brief attacks lasting seconds (60.4% of patients) although the total duration of the vestibular episode lasted hours or days. Other aggravating factors were moving visual stimuli, passive motion, and visually busy environments. Visually induced vestibular symptoms were defined by 71.3% of the patients, and positional motion-induced vestibular symptoms were described by 82.2% of the patients. Vestibular symptoms were mainly defined as feeling the ground slipping from under their feet (40.6%), feeling like there is an earthquake or swaying (27.7%), sensation of rocking on a boat (26.7%), and sensation as if stepping on empty space (24.8%). The majority of the patients (83.2%) previously used vestibular-suppressant drugs, and these drugs were effective temporarily only in 12.9%.
Conclusions: Chronic recurrent dizziness symptoms, rather than internal or external vertigo, are predominant in our VM patients. Recurrent brief dizziness attacks induced upon routine visual and/or postural motion, longstanding symptoms with limited response to vestibular suppressants, and precipitation by typical migraine triggers are suggestive of VM.
PMID: 29017000 [PubMed - as supplied by publisher]
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[Vestibular vertigo in hemispheric strokes].
[Vestibular vertigo in hemispheric strokes].
Zh Nevrol Psikhiatr Im S S Korsakova. 2017;117(8. Vyp. 2):27-31
Authors: Zamergrad MV, Artemev DV, Levin OS
Abstract
The literature on cases of hemispheric stroke manifesting with acute vertigo is reviewed. The authors describe a 56-year-old patient with recurrent acute vestibular syndrome due to hemispheric ischemic stroke. Imaging study revealed ischemic stroke with lesions in a parietal-occipital region of the left hemisphere. Possible mechanisms of vestibular symptoms in hemispheric lesions are discussed.
PMID: 28980578 [PubMed - in process]
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Current Treatment Options: Vestibular Migraine.
| Related Articles |
Current Treatment Options: Vestibular Migraine.
Curr Treat Options Neurol. 2017 Sep 30;19(11):38
Authors: Lauritsen CG, Marmura MJ
Abstract
OPINION STATEMENT: Vestibular migraine (VM) is a disorder with a spectrum of clinical presentations and among the most common causes of chronic vestibular symptoms. Some present with attacks before or during typical migraine, but many others have fluctuating or daily symptoms. While the symptoms and pathogenesis of vestibular migraine may have elements of both central and peripheral disorders, hearing loss should be absent. VM typically worsens with activity and head movements in general, and encompasses symptoms of vertigo, disequilibrium, or imbalance. While it is possible to confuse other disorders such as Meniere's disease with migraine, it is worth making the diagnosis of vestibular migraine on clinical grounds after ruling out other possible causes. For acute attack treatment, migraine-specific medications such as triptans may be effective. In patients with frequent or disabling attacks, preventive treatment is essential. Vestibular rehabilitation may be helpful as well as medications used for migraine prevention such as including propranolol, venlafaxine, topiramate, and amitriptyline. For refractory patients, use of acetazolamide or lamotrigine may be reasonable.
PMID: 28965306 [PubMed]
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21st Annual JDP-LCD Student Colloquium A Huge Success!
The 21st Annual Doctoral Student Colloquium of the SDSU/UCSD Joint Doctoral Program in Language and Communicative Disorders (JDP-LCD), on September 22, 2017, was a big success! Niloofar Akhavan, Phil Combiths, Brittany Lee and Jonathan Robinson Anthony presented research from their 1st- and 2nd- year doctoral projects.
Back row (L-R): Jacob Momsen, Brittany Lee, Niloofar Akhavan, Meghan McGarry, Carolyn Baker
Not shown: Chris Brozdowski, Quynh Dam, Gabriela Meade, Irina Potapova
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