from #Audiology via ola Kala on Inoreader http://ift.tt/2fn5UId
via IFTTT
OtoRhinoLaryngology by Sfakianakis G.Alexandros Sfakianakis G.Alexandros,Anapafseos 5 Agios Nikolaos 72100 Crete Greece,tel : 00302841026182,00306932607174
In this study, piezoelectric cells (PE-cells) of cancellous bone were experimentally produced to receive an ultrasound wave. In the PE-cell, a bovine cancellous bone specimen, in which the pore spaces were saturated with air, was electrically shielded to prevent electromagnetic noise. As a result, the piezoelectric signal generated in the cancellous bone specimen by irradiating an ultrasound burst wave at 1.0 MHz could be clearly observed in water. The experimental results showed that the ultrasound sensitivity per unit area of cancellous bone was estimated to be below 1/100 and 1/100 000 of cortical bone and poly(vinylidene fluoride), respectively.
Source depth estimation with a vertical line array generally involves mode filtering, then matched-mode processing. Because mode filtering is an ill-posed problem if the water column is not well-sampled, concerns for robustness motivate a simpler approach: source depth discrimination considered as a binary classification problem. It aims to evaluate whether the source is near the surface or submerged. These two hypotheses are formulated in terms of normal modes, using the concept of trapped and free modes. Decision metrics based on classic mode filters are proposed. Monte Carlo methods are used to predict performance and set the parameters of a classifier accordingly.
Commentary on the regulatory implications of noise-induced cochlear neuropathy.
Int J Audiol. 2016 Nov 16;:1-5
Authors: Dobie RA, Humes LE
Abstract
OBJECTIVE: A discussion on whether recent research on noise-induced cochlear neuropathy in rodents justifies changes in current regulation of occupational noise exposure.
DESIGN: Informal literature review and commentary, relying on literature found in the authors' files. No formal literature search was performed.
STUDY SAMPLE: Published literature on temporary threshold shift (TTS) and cochlear pathology, in humans and experimental animals, as well as the regulations of the US Occupational Safety and Health Administration (OSHA).
RESULTS: Humans are less susceptible to TTS, and probably to cochlear neuropathy, than rodents. After correcting for inter-species audiometric differences (but not for differences in susceptibility), exposures that caused cochlear neuropathy in rodents already exceed OSHA limits. Those exposures also caused "pathological TTS" (requiring more than 24 h to recover), which does not appear to occur with human broadband noise exposure permissible under OSHA.
CONCLUSION: It would be premature to conclude that noise exposures permissible under OSHA can cause cochlear neuropathy in humans.
PMID: 27849127 [PubMed - as supplied by publisher]
Tinnitus and leisure noise.
Int J Audiol. 2016 Nov 16;:1-7
Authors: Williams W, Carter L
Abstract
OBJECTIVE: To study the relationship of life-time noise exposure and experience of tinnitus.
DESIGN: Audiometric measures included otoscopy, pure tone air- and bone-conduction hearing threshold levels (HTL) and otoacoustic emissions (OAEs). Participants completed questionnaires including demographic information, past hearing health, history of participation in loud leisure activities, and attitudes to noise.
STUDY SAMPLE: A representative sample (1435) of the young (11-35 years old) Australian population.
RESULTS: Of the sample, 63% indicated they experienced tinnitus in some form. There was no correlation of tinnitus experience with HTL or OAE amplitudes. Although median octave band HTLs for those who experienced tinnitus "all the time" were slightly higher for those who did not, neither group exhibited HTLs outside clinically-normal values. Of those who experienced tinnitus a direct correlation was found between frequency of experience of tinnitus and increasing cumulative, life-time noise exposure. Those who experienced tinnitus were more likely to report noticing deterioration in their hearing ability over time and to report difficulty hearing in quiet and/or noisy situations.
CONCLUSIONS: Experience of tinnitus was found throughout this young population but not associated with HTLs or variation in OAE amplitudes. Males experienced 'permanent' tinnitus at significantly greater rate than females.
PMID: 27849126 [PubMed - as supplied by publisher]
Commentary on the regulatory implications of noise-induced cochlear neuropathy.
Int J Audiol. 2016 Nov 16;:1-5
Authors: Dobie RA, Humes LE
Abstract
OBJECTIVE: A discussion on whether recent research on noise-induced cochlear neuropathy in rodents justifies changes in current regulation of occupational noise exposure.
DESIGN: Informal literature review and commentary, relying on literature found in the authors' files. No formal literature search was performed.
STUDY SAMPLE: Published literature on temporary threshold shift (TTS) and cochlear pathology, in humans and experimental animals, as well as the regulations of the US Occupational Safety and Health Administration (OSHA).
RESULTS: Humans are less susceptible to TTS, and probably to cochlear neuropathy, than rodents. After correcting for inter-species audiometric differences (but not for differences in susceptibility), exposures that caused cochlear neuropathy in rodents already exceed OSHA limits. Those exposures also caused "pathological TTS" (requiring more than 24 h to recover), which does not appear to occur with human broadband noise exposure permissible under OSHA.
CONCLUSION: It would be premature to conclude that noise exposures permissible under OSHA can cause cochlear neuropathy in humans.
PMID: 27849127 [PubMed - as supplied by publisher]
Tinnitus and leisure noise.
Int J Audiol. 2016 Nov 16;:1-7
Authors: Williams W, Carter L
Abstract
OBJECTIVE: To study the relationship of life-time noise exposure and experience of tinnitus.
DESIGN: Audiometric measures included otoscopy, pure tone air- and bone-conduction hearing threshold levels (HTL) and otoacoustic emissions (OAEs). Participants completed questionnaires including demographic information, past hearing health, history of participation in loud leisure activities, and attitudes to noise.
STUDY SAMPLE: A representative sample (1435) of the young (11-35 years old) Australian population.
RESULTS: Of the sample, 63% indicated they experienced tinnitus in some form. There was no correlation of tinnitus experience with HTL or OAE amplitudes. Although median octave band HTLs for those who experienced tinnitus "all the time" were slightly higher for those who did not, neither group exhibited HTLs outside clinically-normal values. Of those who experienced tinnitus a direct correlation was found between frequency of experience of tinnitus and increasing cumulative, life-time noise exposure. Those who experienced tinnitus were more likely to report noticing deterioration in their hearing ability over time and to report difficulty hearing in quiet and/or noisy situations.
CONCLUSIONS: Experience of tinnitus was found throughout this young population but not associated with HTLs or variation in OAE amplitudes. Males experienced 'permanent' tinnitus at significantly greater rate than females.
PMID: 27849126 [PubMed - as supplied by publisher]
Commentary on the regulatory implications of noise-induced cochlear neuropathy.
Int J Audiol. 2016 Nov 16;:1-5
Authors: Dobie RA, Humes LE
Abstract
OBJECTIVE: A discussion on whether recent research on noise-induced cochlear neuropathy in rodents justifies changes in current regulation of occupational noise exposure.
DESIGN: Informal literature review and commentary, relying on literature found in the authors' files. No formal literature search was performed.
STUDY SAMPLE: Published literature on temporary threshold shift (TTS) and cochlear pathology, in humans and experimental animals, as well as the regulations of the US Occupational Safety and Health Administration (OSHA).
RESULTS: Humans are less susceptible to TTS, and probably to cochlear neuropathy, than rodents. After correcting for inter-species audiometric differences (but not for differences in susceptibility), exposures that caused cochlear neuropathy in rodents already exceed OSHA limits. Those exposures also caused "pathological TTS" (requiring more than 24 h to recover), which does not appear to occur with human broadband noise exposure permissible under OSHA.
CONCLUSION: It would be premature to conclude that noise exposures permissible under OSHA can cause cochlear neuropathy in humans.
PMID: 27849127 [PubMed - as supplied by publisher]
Tinnitus and leisure noise.
Int J Audiol. 2016 Nov 16;:1-7
Authors: Williams W, Carter L
Abstract
OBJECTIVE: To study the relationship of life-time noise exposure and experience of tinnitus.
DESIGN: Audiometric measures included otoscopy, pure tone air- and bone-conduction hearing threshold levels (HTL) and otoacoustic emissions (OAEs). Participants completed questionnaires including demographic information, past hearing health, history of participation in loud leisure activities, and attitudes to noise.
STUDY SAMPLE: A representative sample (1435) of the young (11-35 years old) Australian population.
RESULTS: Of the sample, 63% indicated they experienced tinnitus in some form. There was no correlation of tinnitus experience with HTL or OAE amplitudes. Although median octave band HTLs for those who experienced tinnitus "all the time" were slightly higher for those who did not, neither group exhibited HTLs outside clinically-normal values. Of those who experienced tinnitus a direct correlation was found between frequency of experience of tinnitus and increasing cumulative, life-time noise exposure. Those who experienced tinnitus were more likely to report noticing deterioration in their hearing ability over time and to report difficulty hearing in quiet and/or noisy situations.
CONCLUSIONS: Experience of tinnitus was found throughout this young population but not associated with HTLs or variation in OAE amplitudes. Males experienced 'permanent' tinnitus at significantly greater rate than females.
PMID: 27849126 [PubMed - as supplied by publisher]
Commentary on the regulatory implications of noise-induced cochlear neuropathy.
Int J Audiol. 2016 Nov 16;:1-5
Authors: Dobie RA, Humes LE
Abstract
OBJECTIVE: A discussion on whether recent research on noise-induced cochlear neuropathy in rodents justifies changes in current regulation of occupational noise exposure.
DESIGN: Informal literature review and commentary, relying on literature found in the authors' files. No formal literature search was performed.
STUDY SAMPLE: Published literature on temporary threshold shift (TTS) and cochlear pathology, in humans and experimental animals, as well as the regulations of the US Occupational Safety and Health Administration (OSHA).
RESULTS: Humans are less susceptible to TTS, and probably to cochlear neuropathy, than rodents. After correcting for inter-species audiometric differences (but not for differences in susceptibility), exposures that caused cochlear neuropathy in rodents already exceed OSHA limits. Those exposures also caused "pathological TTS" (requiring more than 24 h to recover), which does not appear to occur with human broadband noise exposure permissible under OSHA.
CONCLUSION: It would be premature to conclude that noise exposures permissible under OSHA can cause cochlear neuropathy in humans.
PMID: 27849127 [PubMed - as supplied by publisher]
Tinnitus and leisure noise.
Int J Audiol. 2016 Nov 16;:1-7
Authors: Williams W, Carter L
Abstract
OBJECTIVE: To study the relationship of life-time noise exposure and experience of tinnitus.
DESIGN: Audiometric measures included otoscopy, pure tone air- and bone-conduction hearing threshold levels (HTL) and otoacoustic emissions (OAEs). Participants completed questionnaires including demographic information, past hearing health, history of participation in loud leisure activities, and attitudes to noise.
STUDY SAMPLE: A representative sample (1435) of the young (11-35 years old) Australian population.
RESULTS: Of the sample, 63% indicated they experienced tinnitus in some form. There was no correlation of tinnitus experience with HTL or OAE amplitudes. Although median octave band HTLs for those who experienced tinnitus "all the time" were slightly higher for those who did not, neither group exhibited HTLs outside clinically-normal values. Of those who experienced tinnitus a direct correlation was found between frequency of experience of tinnitus and increasing cumulative, life-time noise exposure. Those who experienced tinnitus were more likely to report noticing deterioration in their hearing ability over time and to report difficulty hearing in quiet and/or noisy situations.
CONCLUSIONS: Experience of tinnitus was found throughout this young population but not associated with HTLs or variation in OAE amplitudes. Males experienced 'permanent' tinnitus at significantly greater rate than females.
PMID: 27849126 [PubMed - as supplied by publisher]