Open Versus Closed Hearing-Aid Fittings: A Literature Review of Both Fitting Approaches

One of the main issues in hearing-aid fittings is the abnormal perception of the user’s own voice as too loud, "boomy," or "hollow." This phenomenon known as the occlusion effect be reduced by large vents in the earmolds or by open-fit hearing aids. This review provides an overview of publications related to open and closed hearing-aid fittings. First, the occlusion effect and its consequences for perception while using hearing aids are described. Then, the advantages and disadvantages of open compared with closed fittings and their impact on the fitting process are addressed. The advantages include less occlusion, improved own-voice perception and sound quality, and increased localization performance. The disadvantages associated with open-fit hearing aids include reduced benefits of directional microphones and noise reduction, as well as less compression and less available gain before feedback. The final part of this review addresses the need for new approaches to combine the advantages of open and closed hearing-aid fittings.

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Open Versus Closed Hearing-Aid Fittings: A Literature Review of Both Fitting Approaches

One of the main issues in hearing-aid fittings is the abnormal perception of the user’s own voice as too loud, "boomy," or "hollow." This phenomenon known as the occlusion effect be reduced by large vents in the earmolds or by open-fit hearing aids. This review provides an overview of publications related to open and closed hearing-aid fittings. First, the occlusion effect and its consequences for perception while using hearing aids are described. Then, the advantages and disadvantages of open compared with closed fittings and their impact on the fitting process are addressed. The advantages include less occlusion, improved own-voice perception and sound quality, and increased localization performance. The disadvantages associated with open-fit hearing aids include reduced benefits of directional microphones and noise reduction, as well as less compression and less available gain before feedback. The final part of this review addresses the need for new approaches to combine the advantages of open and closed hearing-aid fittings.

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Open Versus Closed Hearing-Aid Fittings: A Literature Review of Both Fitting Approaches

One of the main issues in hearing-aid fittings is the abnormal perception of the user’s own voice as too loud, "boomy," or "hollow." This phenomenon known as the occlusion effect be reduced by large vents in the earmolds or by open-fit hearing aids. This review provides an overview of publications related to open and closed hearing-aid fittings. First, the occlusion effect and its consequences for perception while using hearing aids are described. Then, the advantages and disadvantages of open compared with closed fittings and their impact on the fitting process are addressed. The advantages include less occlusion, improved own-voice perception and sound quality, and increased localization performance. The disadvantages associated with open-fit hearing aids include reduced benefits of directional microphones and noise reduction, as well as less compression and less available gain before feedback. The final part of this review addresses the need for new approaches to combine the advantages of open and closed hearing-aid fittings.

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Vagus Nerve Stimulation For Tinnitus

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People suffering from severe tinnitus have a hard time finding helpful therapies. Now, a new treatment using vagus nerve stimulation for tinnitus relief is showing promising results for many people with this frustrating and debilitating condition.

Tinnitus is a medical condition in which a person hears ringing, whooshing, or buzzing sounds inside their head and ears. Many people experience short episodes of tinnitus symptoms from time to time. People with bad cases of tinnitus hear these sounds all the time, causing severe distraction, discomfort, and annoyance.

The Wandering Vagus Nerve

The vagus nerve is actually a long pair of nerves wandering throughout the body. (The origin of the word vagus means wanderer.) Much of the work done by the vagus nerve involves sending messages from bodily organs to the brain, alerting the brain about what the body is experiencing.

The vagus nerve is part of the parasympathetic nervous system starting in the 10th cranial nerve in the spinal cord and extending to the heart, stomach and other organs, and into the brain. The parasympathetic nervous system is composed of the nerves and hormonal receptors which quiet the body down after ‘fight or flight’ messages from the sympathetic nervous system.

Artificial stimulation of the vagus nerve has been in use for many years by researchers and medical professionals as a treatment for severe epilepsy. Now, new technology has been developed for stimulating the vagus nerve for tinnitus treatment, and many tinnitus sufferers are finding help with this therapy.

How Vagus Nerve Stimulation for Tinnitus Works

This therapy for tinnitus works by implanting a small electrical stimulator into the patient’s neck. The devise attaches to the vagus nerve close to the point where it enters the auditory cortex. The patient then puts on a set of headphones and listens to a series of tones specifically designed to train their brain to ignore the tinnitus frequencies.

During a session of using vagus nerve stimulation for tinnitus therapy, the patient hears frequencies above and below the frequency of the bothersome tinnitus tones. Over time, the person’s brain learns to ignore the tinnitus sounds, restoring normal activity in the auditory cortex.

For many people using vagus nerve stimulation for tinnitus treatment, the bothersome tinnitus sounds go away or subside considerably for several weeks after a treatment session. The treatment can then be repeated as needed in the patient’s home using headphones and the digital audio application.

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Factors influencing pursuit of hearing evaluation: Enhancing the health belief model with perceived burden from hearing loss on communication partners

10.3109/14992027.2015.1136437<br/>Kristine A. Schulz

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Predicting three-month and 12-month post-fitting real-world hearing-aid outcome using pre-fitting acceptable noise level (ANL)

10.3109/14992027.2015.1120892<br/>Yu-Hsiang Wu

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Factors influencing pursuit of hearing evaluation: Enhancing the health belief model with perceived burden from hearing loss on communication partners

10.3109/14992027.2015.1136437<br/>Kristine A. Schulz

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Predicting three-month and 12-month post-fitting real-world hearing-aid outcome using pre-fitting acceptable noise level (ANL)

10.3109/14992027.2015.1120892<br/>Yu-Hsiang Wu

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Factors influencing pursuit of hearing evaluation: Enhancing the health belief model with perceived burden from hearing loss on communication partners

10.3109/14992027.2015.1136437<br/>Kristine A. Schulz

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Predicting three-month and 12-month post-fitting real-world hearing-aid outcome using pre-fitting acceptable noise level (ANL)

10.3109/14992027.2015.1120892<br/>Yu-Hsiang Wu

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Electrode array-eluted dexamethasone protects against electrode insertion trauma induced hearing and hair cell losses, damage to neural elements, increases in impedance and fibrosis: A dose response study

Publication date: Available online 15 February 2016
Source:Hearing Research
Author(s): Esperanza Bas, Jorge Bohorquez, Stefania Goncalves, Enrique Perez, Christine T. Dinh, Carolyn Garnham, Roland Hessler, Adrien A. Eshraghi, Thomas R. Van De Water
We evaluated the effects of dexamethasone base (DXMb) containing electrode arrays in a guinea pig model of cochlear implantation to determine if eluted DXMb could protect the cochlea against electrode insertion trauma (EIT)-induced: 1) loss of hair cells; 2) disruption of neural elements; 3) increases in hearing thresholds; 4) increased electrical impedance and 5) fibrosis. A guinea pig model of EIT-induced hearing and hair cell losses was used to test silicone electrode arrays that contained either 10%, 1%, 0.1%, or 0% levels of micronized DXMb. These four types of electrode arrays were implanted into the scala tympani via basal turn cochleostomies and left in place for 3 months. Hearing thresholds were determined by ABR and CAP recordings in response to a series of defined pure tone stimuli (i.e. 16 to 0.5 kHz). Changes in impedance were measured between the implant electrode and a reference electrode. Hair cell counts and neural element integrity were determined by confocal microscopy analyses of stained organ of Corti whole mounts obtained from 90 day post-implantation animals. Fibrosis was measured in Masson trichrome stained cross-sections through the organ of Corti. The results showed that either 10% or 1.0% DXMb eluting electrode arrays protected; hearing thresholds, hair cells, and neural elements against EIT-induced losses and damage. Electrode arrays with 0.1% DXMb only partial protected against EIT-induced hearing loss and damage to the cochlea. Protection of hearing thresholds and organ of Corti sensory elements by electrode-eluted DXMb was still apparent at 3 months post-EIT. All three concentrations of DXMb in the electrode arrays prevented EIT-induced increases in impedance. EIT-initiated fibrosis was significantly reduced within the implanted cochlea of the two DXMb concentrations tested. In conclusion, DXMb eluting electrodes protected the cochlea against long term increases in hearing thresholds, loss of hair cells, damage to neural elements and increases in impedance and fibrosis that result from EIT-initiated damage. The protection achieved by DXMb-eluting electrodes was dose dependent. Establishing a significant level of trauma induced elevation in hearing thresholds was important for the determination of the otoprotective effects of array-eluted DXMb.

Graphical abstract

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Electrode array-eluted dexamethasone protects against electrode insertion trauma induced hearing and hair cell losses, damage to neural elements, increases in impedance and fibrosis: A dose response study

Publication date: Available online 15 February 2016
Source:Hearing Research
Author(s): Esperanza Bas, Jorge Bohorquez, Stefania Goncalves, Enrique Perez, Christine T. Dinh, Carolyn Garnham, Roland Hessler, Adrien A. Eshraghi, Thomas R. Van De Water
We evaluated the effects of dexamethasone base (DXMb) containing electrode arrays in a guinea pig model of cochlear implantation to determine if eluted DXMb could protect the cochlea against electrode insertion trauma (EIT)-induced: 1) loss of hair cells; 2) disruption of neural elements; 3) increases in hearing thresholds; 4) increased electrical impedance and 5) fibrosis. A guinea pig model of EIT-induced hearing and hair cell losses was used to test silicone electrode arrays that contained either 10%, 1%, 0.1%, or 0% levels of micronized DXMb. These four types of electrode arrays were implanted into the scala tympani via basal turn cochleostomies and left in place for 3 months. Hearing thresholds were determined by ABR and CAP recordings in response to a series of defined pure tone stimuli (i.e. 16 to 0.5 kHz). Changes in impedance were measured between the implant electrode and a reference electrode. Hair cell counts and neural element integrity were determined by confocal microscopy analyses of stained organ of Corti whole mounts obtained from 90 day post-implantation animals. Fibrosis was measured in Masson trichrome stained cross-sections through the organ of Corti. The results showed that either 10% or 1.0% DXMb eluting electrode arrays protected; hearing thresholds, hair cells, and neural elements against EIT-induced losses and damage. Electrode arrays with 0.1% DXMb only partial protected against EIT-induced hearing loss and damage to the cochlea. Protection of hearing thresholds and organ of Corti sensory elements by electrode-eluted DXMb was still apparent at 3 months post-EIT. All three concentrations of DXMb in the electrode arrays prevented EIT-induced increases in impedance. EIT-initiated fibrosis was significantly reduced within the implanted cochlea of the two DXMb concentrations tested. In conclusion, DXMb eluting electrodes protected the cochlea against long term increases in hearing thresholds, loss of hair cells, damage to neural elements and increases in impedance and fibrosis that result from EIT-initiated damage. The protection achieved by DXMb-eluting electrodes was dose dependent. Establishing a significant level of trauma induced elevation in hearing thresholds was important for the determination of the otoprotective effects of array-eluted DXMb.

Graphical abstract

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Electrode array-eluted dexamethasone protects against electrode insertion trauma induced hearing and hair cell losses, damage to neural elements, increases in impedance and fibrosis: A dose response study

Publication date: Available online 15 February 2016
Source:Hearing Research
Author(s): Esperanza Bas, Jorge Bohorquez, Stefania Goncalves, Enrique Perez, Christine T. Dinh, Carolyn Garnham, Roland Hessler, Adrien A. Eshraghi, Thomas R. Van De Water
We evaluated the effects of dexamethasone base (DXMb) containing electrode arrays in a guinea pig model of cochlear implantation to determine if eluted DXMb could protect the cochlea against electrode insertion trauma (EIT)-induced: 1) loss of hair cells; 2) disruption of neural elements; 3) increases in hearing thresholds; 4) increased electrical impedance and 5) fibrosis. A guinea pig model of EIT-induced hearing and hair cell losses was used to test silicone electrode arrays that contained either 10%, 1%, 0.1%, or 0% levels of micronized DXMb. These four types of electrode arrays were implanted into the scala tympani via basal turn cochleostomies and left in place for 3 months. Hearing thresholds were determined by ABR and CAP recordings in response to a series of defined pure tone stimuli (i.e. 16 to 0.5 kHz). Changes in impedance were measured between the implant electrode and a reference electrode. Hair cell counts and neural element integrity were determined by confocal microscopy analyses of stained organ of Corti whole mounts obtained from 90 day post-implantation animals. Fibrosis was measured in Masson trichrome stained cross-sections through the organ of Corti. The results showed that either 10% or 1.0% DXMb eluting electrode arrays protected; hearing thresholds, hair cells, and neural elements against EIT-induced losses and damage. Electrode arrays with 0.1% DXMb only partial protected against EIT-induced hearing loss and damage to the cochlea. Protection of hearing thresholds and organ of Corti sensory elements by electrode-eluted DXMb was still apparent at 3 months post-EIT. All three concentrations of DXMb in the electrode arrays prevented EIT-induced increases in impedance. EIT-initiated fibrosis was significantly reduced within the implanted cochlea of the two DXMb concentrations tested. In conclusion, DXMb eluting electrodes protected the cochlea against long term increases in hearing thresholds, loss of hair cells, damage to neural elements and increases in impedance and fibrosis that result from EIT-initiated damage. The protection achieved by DXMb-eluting electrodes was dose dependent. Establishing a significant level of trauma induced elevation in hearing thresholds was important for the determination of the otoprotective effects of array-eluted DXMb.

Graphical abstract

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Electrode array-eluted dexamethasone protects against electrode insertion trauma induced hearing and hair cell losses, damage to neural elements, increases in impedance and fibrosis: A dose response study

Publication date: Available online 15 February 2016
Source:Hearing Research
Author(s): Esperanza Bas, Jorge Bohorquez, Stefania Goncalves, Enrique Perez, Christine T. Dinh, Carolyn Garnham, Roland Hessler, Adrien A. Eshraghi, Thomas R. Van De Water
We evaluated the effects of dexamethasone base (DXMb) containing electrode arrays in a guinea pig model of cochlear implantation to determine if eluted DXMb could protect the cochlea against electrode insertion trauma (EIT)-induced: 1) loss of hair cells; 2) disruption of neural elements; 3) increases in hearing thresholds; 4) increased electrical impedance and 5) fibrosis. A guinea pig model of EIT-induced hearing and hair cell losses was used to test silicone electrode arrays that contained either 10%, 1%, 0.1%, or 0% levels of micronized DXMb. These four types of electrode arrays were implanted into the scala tympani via basal turn cochleostomies and left in place for 3 months. Hearing thresholds were determined by ABR and CAP recordings in response to a series of defined pure tone stimuli (i.e. 16 to 0.5 kHz). Changes in impedance were measured between the implant electrode and a reference electrode. Hair cell counts and neural element integrity were determined by confocal microscopy analyses of stained organ of Corti whole mounts obtained from 90 day post-implantation animals. Fibrosis was measured in Masson trichrome stained cross-sections through the organ of Corti. The results showed that either 10% or 1.0% DXMb eluting electrode arrays protected; hearing thresholds, hair cells, and neural elements against EIT-induced losses and damage. Electrode arrays with 0.1% DXMb only partial protected against EIT-induced hearing loss and damage to the cochlea. Protection of hearing thresholds and organ of Corti sensory elements by electrode-eluted DXMb was still apparent at 3 months post-EIT. All three concentrations of DXMb in the electrode arrays prevented EIT-induced increases in impedance. EIT-initiated fibrosis was significantly reduced within the implanted cochlea of the two DXMb concentrations tested. In conclusion, DXMb eluting electrodes protected the cochlea against long term increases in hearing thresholds, loss of hair cells, damage to neural elements and increases in impedance and fibrosis that result from EIT-initiated damage. The protection achieved by DXMb-eluting electrodes was dose dependent. Establishing a significant level of trauma induced elevation in hearing thresholds was important for the determination of the otoprotective effects of array-eluted DXMb.

Graphical abstract

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Electrode array-eluted dexamethasone protects against electrode insertion trauma induced hearing and hair cell losses, damage to neural elements, increases in impedance and fibrosis: A dose response study

Publication date: Available online 15 February 2016
Source:Hearing Research
Author(s): Esperanza Bas, Jorge Bohorquez, Stefania Goncalves, Enrique Perez, Christine T. Dinh, Carolyn Garnham, Roland Hessler, Adrien A. Eshraghi, Thomas R. Van De Water
We evaluated the effects of dexamethasone base (DXMb) containing electrode arrays in a guinea pig model of cochlear implantation to determine if eluted DXMb could protect the cochlea against electrode insertion trauma (EIT)-induced: 1) loss of hair cells; 2) disruption of neural elements; 3) increases in hearing thresholds; 4) increased electrical impedance and 5) fibrosis. A guinea pig model of EIT-induced hearing and hair cell losses was used to test silicone electrode arrays that contained either 10%, 1%, 0.1%, or 0% levels of micronized DXMb. These four types of electrode arrays were implanted into the scala tympani via basal turn cochleostomies and left in place for 3 months. Hearing thresholds were determined by ABR and CAP recordings in response to a series of defined pure tone stimuli (i.e. 16 to 0.5 kHz). Changes in impedance were measured between the implant electrode and a reference electrode. Hair cell counts and neural element integrity were determined by confocal microscopy analyses of stained organ of Corti whole mounts obtained from 90 day post-implantation animals. Fibrosis was measured in Masson trichrome stained cross-sections through the organ of Corti. The results showed that either 10% or 1.0% DXMb eluting electrode arrays protected; hearing thresholds, hair cells, and neural elements against EIT-induced losses and damage. Electrode arrays with 0.1% DXMb only partial protected against EIT-induced hearing loss and damage to the cochlea. Protection of hearing thresholds and organ of Corti sensory elements by electrode-eluted DXMb was still apparent at 3 months post-EIT. All three concentrations of DXMb in the electrode arrays prevented EIT-induced increases in impedance. EIT-initiated fibrosis was significantly reduced within the implanted cochlea of the two DXMb concentrations tested. In conclusion, DXMb eluting electrodes protected the cochlea against long term increases in hearing thresholds, loss of hair cells, damage to neural elements and increases in impedance and fibrosis that result from EIT-initiated damage. The protection achieved by DXMb-eluting electrodes was dose dependent. Establishing a significant level of trauma induced elevation in hearing thresholds was important for the determination of the otoprotective effects of array-eluted DXMb.

Graphical abstract

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Is proprioception diminished in patients with patellar tendinopathy?

Publication date: Available online 15 February 2016
Source:Gait & Posture
Author(s): H.E. Groot, H. van der Worp, L. Nijenbanning, R.L. Diercks, J. Zwerver, I. van den Akker-Scheek
PurposePatellar tendinopathy is a highly prevalent overuse injury, and most treatments are only effective to some extent. This persistence of complaints could be linked to changed proprioception. One study showed diminished proprioception in athletes with lateral epicondylitis. Aim of this study was to determine differences in proprioception, by measuring threshold to detect passive motion (TTDPM) between recreational athletes diagnosed with patellar tendinopathy and healthy controls.MethodThe TTDPM as measure of proprioception was determined in 22 recreational athletes with patellar tendinopathy and 22 healthy recreational athletes using a validated instrument. Amount of knee flexion and extension before the movement was noticed by the subject was determined. 80 measurements per athlete (left and right leg, towards extension and flexion and with two starting angles of 20° and 40° flexion) were performed. Mean TTDPM was compared between groups and among the injured recreational athletes between the affected and unaffected knee.ResultsNo significant difference in TTDPM was found between recreational athletes with patellar tendinopathy and healthy controls. We did find a significant difference between the injured and non-injured knee in recreational athletes with patellar tendinopathy; mean TTDPM was 0.02° higher in the injured knee (p=0.044).ConclusionNo difference was found in proprioception between recreational athletes with patellar tendinopathy and healthy recreational athletes. It is unclear whether such a small difference in TTDPM between affected and unaffected knee is important in clinical setting.



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Is proprioception diminished in patients with patellar tendinopathy?

Publication date: Available online 15 February 2016
Source:Gait & Posture
Author(s): H.E. Groot, H. van der Worp, L. Nijenbanning, R.L. Diercks, J. Zwerver, I. van den Akker-Scheek
PurposePatellar tendinopathy is a highly prevalent overuse injury, and most treatments are only effective to some extent. This persistence of complaints could be linked to changed proprioception. One study showed diminished proprioception in athletes with lateral epicondylitis. Aim of this study was to determine differences in proprioception, by measuring threshold to detect passive motion (TTDPM) between recreational athletes diagnosed with patellar tendinopathy and healthy controls.MethodThe TTDPM as measure of proprioception was determined in 22 recreational athletes with patellar tendinopathy and 22 healthy recreational athletes using a validated instrument. Amount of knee flexion and extension before the movement was noticed by the subject was determined. 80 measurements per athlete (left and right leg, towards extension and flexion and with two starting angles of 20° and 40° flexion) were performed. Mean TTDPM was compared between groups and among the injured recreational athletes between the affected and unaffected knee.ResultsNo significant difference in TTDPM was found between recreational athletes with patellar tendinopathy and healthy controls. We did find a significant difference between the injured and non-injured knee in recreational athletes with patellar tendinopathy; mean TTDPM was 0.02° higher in the injured knee (p=0.044).ConclusionNo difference was found in proprioception between recreational athletes with patellar tendinopathy and healthy recreational athletes. It is unclear whether such a small difference in TTDPM between affected and unaffected knee is important in clinical setting.



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via IFTTT

Is proprioception diminished in patients with patellar tendinopathy?

Publication date: Available online 15 February 2016
Source:Gait & Posture
Author(s): H.E. Groot, H. van der Worp, L. Nijenbanning, R.L. Diercks, J. Zwerver, I. van den Akker-Scheek
PurposePatellar tendinopathy is a highly prevalent overuse injury, and most treatments are only effective to some extent. This persistence of complaints could be linked to changed proprioception. One study showed diminished proprioception in athletes with lateral epicondylitis. Aim of this study was to determine differences in proprioception, by measuring threshold to detect passive motion (TTDPM) between recreational athletes diagnosed with patellar tendinopathy and healthy controls.MethodThe TTDPM as measure of proprioception was determined in 22 recreational athletes with patellar tendinopathy and 22 healthy recreational athletes using a validated instrument. Amount of knee flexion and extension before the movement was noticed by the subject was determined. 80 measurements per athlete (left and right leg, towards extension and flexion and with two starting angles of 20° and 40° flexion) were performed. Mean TTDPM was compared between groups and among the injured recreational athletes between the affected and unaffected knee.ResultsNo significant difference in TTDPM was found between recreational athletes with patellar tendinopathy and healthy controls. We did find a significant difference between the injured and non-injured knee in recreational athletes with patellar tendinopathy; mean TTDPM was 0.02° higher in the injured knee (p=0.044).ConclusionNo difference was found in proprioception between recreational athletes with patellar tendinopathy and healthy recreational athletes. It is unclear whether such a small difference in TTDPM between affected and unaffected knee is important in clinical setting.



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Introducing the New Widex UNIQUE: Because Every Situation is Unique

Widex has a long legacy, for a number of reasons. Number one is sound quality and the ease of listening. Those of you who have worked with Widex know what our hearing aids sound like. If you are new to Widex, we welcome you to try our product, and you will learn that there is a specific Widex sound; our number one goal is ease of listening. The goal of the engineers at Widex is to make sound as close to natural as possible. We are true to audiology. We are true to the principles of hearing health care. We preserve audibility; it underlies our features, our algorithms, and prescriptions.

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New RECDs and a New ANSI Standard: Revisiting RECD Basics and Applications

Dr. Susan Scollie: Verification of hearing aids is best practice, because we know that people of all ages vary in their ear canal acoustics (Bagatto et al., 2002; Saunders & Morgan, 2003). If we don't account for that variability, then some people's hearing aids could be too loud or too soft, and that might impact either their comfort and/or their benefit from the devices (Aarts & Caffee, 2005; Aazh & Moore, 2007; Kochkin, et al., 2010; McCreery, Bentler, & Roush, 2013). Hearing aid fittings should be individualized to protect against these large errors and ensure consistent audibility of speech (Mueller, 2014).

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Introducing the New Widex UNIQUE: Because Every Situation is Unique

Widex has a long legacy, for a number of reasons. Number one is sound quality and the ease of listening. Those of you who have worked with Widex know what our hearing aids sound like. If you are new to Widex, we welcome you to try our product, and you will learn that there is a specific Widex sound; our number one goal is ease of listening. The goal of the engineers at Widex is to make sound as close to natural as possible. We are true to audiology. We are true to the principles of hearing health care. We preserve audibility; it underlies our features, our algorithms, and prescriptions.

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New RECDs and a New ANSI Standard: Revisiting RECD Basics and Applications

Dr. Susan Scollie: Verification of hearing aids is best practice, because we know that people of all ages vary in their ear canal acoustics (Bagatto et al., 2002; Saunders & Morgan, 2003). If we don't account for that variability, then some people's hearing aids could be too loud or too soft, and that might impact either their comfort and/or their benefit from the devices (Aarts & Caffee, 2005; Aazh & Moore, 2007; Kochkin, et al., 2010; McCreery, Bentler, & Roush, 2013). Hearing aid fittings should be individualized to protect against these large errors and ensure consistent audibility of speech (Mueller, 2014).

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via IFTTT

Introducing the New Widex UNIQUE: Because Every Situation is Unique

Widex has a long legacy, for a number of reasons. Number one is sound quality and the ease of listening. Those of you who have worked with Widex know what our hearing aids sound like. If you are new to Widex, we welcome you to try our product, and you will learn that there is a specific Widex sound; our number one goal is ease of listening. The goal of the engineers at Widex is to make sound as close to natural as possible. We are true to audiology. We are true to the principles of hearing health care. We preserve audibility; it underlies our features, our algorithms, and prescriptions.

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via IFTTT

New RECDs and a New ANSI Standard: Revisiting RECD Basics and Applications

Dr. Susan Scollie: Verification of hearing aids is best practice, because we know that people of all ages vary in their ear canal acoustics (Bagatto et al., 2002; Saunders & Morgan, 2003). If we don't account for that variability, then some people's hearing aids could be too loud or too soft, and that might impact either their comfort and/or their benefit from the devices (Aarts & Caffee, 2005; Aazh & Moore, 2007; Kochkin, et al., 2010; McCreery, Bentler, & Roush, 2013). Hearing aid fittings should be individualized to protect against these large errors and ensure consistent audibility of speech (Mueller, 2014).

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